PMID 42004906

Research
All papers

Effect of high-intensity interval training on peak oxygen uptake, quality of life, and ventricular arrhythmias in patients with an implantable cardioverter defibrillator: a randomized controlled trial

Question

Does a supervised 12-week HIIT programme improve exercise capacity and quality of life, and how does it affect ventricular arrhythmia burden, in selected ICD/CRT-D patients with coronary artery disease or non-ischaemic dilated cardiomyopathy?

Summary

In a single-centre randomized trial, 56 adults with an implantable cardioverter defibrillator or cardiac resynchronization therapy defibrillator because of coronary artery disease or non-ischaemic dilated cardiomyopathy were assigned to 12 weeks of supervised treadmill HIIT or usual activity. The HIIT protocol used 4 x 4 minute intervals at 85-95% of maximum heart rate, three times per week. HIIT improved VO2peak by about 7% compared with no change in controls and improved the SF-36 health change quality-of-life domain after multiple-comparison correction. Arrhythmia findings were inconclusive: sustained ventricular tachycardia incidence was statistically lower in the HIIT group, but events were few and unevenly distributed, and two HIIT participants had sustained ventricular tachycardia during training.

Methodology

  • Selected adults with ICD or CRT-D for coronary artery disease or non-ischaemic dilated cardiomyopathy.
  • 56 participants.
  • Uphill treadmill walking/running.
  • Work intervals: 4 minutes at 85-95% HRmax and/or Borg RPE 15-17.
  • Recovery: 3 minutes active recovery at 60-70% HRmax and/or Borg RPE 11-13.
  • Intensity: Prescribed 85-95% HRmax/RPE 15-17 during intervals.
  • 40 minutes.
  • 3 sessions/week.
  • 12 weeks.
  • Single-centre, parallel-group randomized controlled trial with stratification by coronary artery disease versus dilated cardiomyopathy and 1:1 allocation to supervised HIIT or usual activity control.
  • VO2peak, Quality of life, Ventricular arrhythmia burden, and Premature ventricular contractions and VT by Holter were tracked.

Outcomes

VO2peak

HIIT increased VO2peak by 1.6 +/- 2.1 mL/kg/min (7.0%) from 24.4 to 26.1 mL/kg/min, while controls had no change.

Between-group difference 1.7 mL/kg/min, 95% CI 0.7 to 2.6; P < 0.001.

Improved

SF-36 health change

The SF-36 health change subscale improved in the HIIT group and declined in controls.

+14.8 +/- 22.2 points after HIIT versus -7.1 +/- 23.4 in controls; 95% CI 9.6 to 34.3; P < 0.001 after Bonferroni correction.

Improved

Other SF-36 domains

Most other quality-of-life domains showed favourable numerical trends but did not remain statistically significant after correction.

Bonferroni-adjusted alpha was 0.006 across nine subscales.

Mixed

Sustained ventricular tachycardia incidence

Sustained VT incidence decreased in HIIT and increased in controls from baseline to follow-up, but events were few and unevenly distributed.

Group difference -1.8 events per patient-year at follow-up; P = 0.037.

Mixed

Exercise-related adverse events

Two HIIT participants had sustained VT during training, including one ICD shock after VT accelerated to ventricular fibrillation with loss of consciousness; one participant had a knee injury during training.

Safety concern

Insights

  • A classic 4 x 4 minute HIIT structure can improve VO2peak in selected cardiac rehabilitation patients when supervised and intensity-monitored.
  • In high-risk ICD populations, HIIT programming must be tied to clinical screening, emergency readiness, and individualized risk evaluation.
  • RPE can complement heart-rate targets when beta-blockers and device settings affect heart-rate interpretation.
  • Consumer-facing HIIT guidance should avoid implying that ICD patients can perform high-intensity intervals without medical clearance.

Limitations

  • Single-centre study.
  • Small sample and under-recruitment relative to planned sample size.
  • Short intervention duration.
  • Not powered for safety outcomes.
  • Low inclusion rate and possible selection/volunteer bias.
  • Women underrepresented.
  • Baseline imbalances may have affected clinical and arrhythmia outcomes.
  • Participants and exercise staff could not be blinded to exercise intervention.
  • Control group activity was not actively monitored and some control contamination occurred.
  • Excluded important ICD subgroups and patients with exercise-induced arrhythmia at baseline.

Safety

  • Clinically relevant ventricular arrhythmias occurred in two patients during baseline CPET and two patients during HIIT.
  • Two sustained VT events occurred during HIIT; one required ICD shock and one was treated with ATP.
  • One knee injury during training led to withdrawal.
  • Several adverse events unrelated to exercise were reported, including ICD lead issues, angina/revascularization in control, gastrointestinal bleeding in control, subdural haemorrhage after a fall, and atrial flutter hospitalization.